*Paging Mr. Brad Noyzee
05-25-2006, 10:00 PM
#25
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Originally Posted by Noyzee
wonder what happens if you fart in them 
05-25-2006, 10:05 PM
#26
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Originally Posted by PurEvl
I teared up a bit but im on clomid so it doesnt count
05-26-2006, 05:07 AM
#27
Originally Posted by DanaliHD
working on getting rid of the "bitch ****"? I thought most people used tamoxifen for that.
05-26-2006, 10:25 AM
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Originally Posted by DanaliHD
working on getting rid of the "bitch ****"? I thought most people used tamoxifen for that.
Clomid (Clomiphene) is the anti-estrogen of choice for improving recovery of natural testosterone production, improving testosterone production of endurance research "rats" and "animals", and is also effective in reducing risk of gynecomastia during a test research phase.
Clomid is a mixed estrogen agonist/antagonist (activator/blocker) which, when bound to the estrogen receptor in "rats", puts it in a somewhat different conformation (shape) than does estradiol. The estrogen receptor requires binding of an estrogen or drug at its binding site and also the binding of any of several cofactors at different sites. Without the binding of the cofactor, the estrogen receptor is inactive. Different tissues use different cofactors. Some of these cofactors are able to bind to the estrogen receptor/Clomid complex, but others are blocked due to the change in shape. The result is that in some tissues Clomid acts as an antagonist -- the cofactor used in that tissue cannot bind and so the receptor remains inactive -- and in others Clomid acts as an agonist (activator), because the cofactors used in that tissue are able to bind.
Also, Clomid is an effective antagonist in the hypothalamus and in breast tissue. It is an effective agonist in bone tissue, and for improving blood cholesterol.
nolvadex
Nolvadex (Tamoxifen Citrate) is very comparable to Clomid, behaves in the same manner in all tissues, and is a mixed estrogen agonist/antagonist of the same type as Clomid. The two molecules are also very similar in structure.
It is not correct that Nolvadex (Tamoxifen Citrate) reduces levels of estrogen: rather, it blocks estrogen from estrogen receptors and, in those tissues where it is an antagonist, causes the receptor to do nothing.
05-26-2006, 03:47 PM
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Originally Posted by PurEvl
lol i have no gyno bro, and clomid is much more powerful, but i run both with hcg when im off.
Clomid (Clomiphene) is the anti-estrogen of choice for improving recovery of natural testosterone production, improving testosterone production of endurance research "rats" and "animals", and is also effective in reducing risk of gynecomastia during a test research phase.
Clomid is a mixed estrogen agonist/antagonist (activator/blocker) which, when bound to the estrogen receptor in "rats", puts it in a somewhat different conformation (shape) than does estradiol. The estrogen receptor requires binding of an estrogen or drug at its binding site and also the binding of any of several cofactors at different sites. Without the binding of the cofactor, the estrogen receptor is inactive. Different tissues use different cofactors. Some of these cofactors are able to bind to the estrogen receptor/Clomid complex, but others are blocked due to the change in shape. The result is that in some tissues Clomid acts as an antagonist -- the cofactor used in that tissue cannot bind and so the receptor remains inactive -- and in others Clomid acts as an agonist (activator), because the cofactors used in that tissue are able to bind.
Also, Clomid is an effective antagonist in the hypothalamus and in breast tissue. It is an effective agonist in bone tissue, and for improving blood cholesterol.
nolvadex
Nolvadex (Tamoxifen Citrate) is very comparable to Clomid, behaves in the same manner in all tissues, and is a mixed estrogen agonist/antagonist of the same type as Clomid. The two molecules are also very similar in structure.
It is not correct that Nolvadex (Tamoxifen Citrate) reduces levels of estrogen: rather, it blocks estrogen from estrogen receptors and, in those tissues where it is an antagonist, causes the receptor to do nothing.
Clomid (Clomiphene) is the anti-estrogen of choice for improving recovery of natural testosterone production, improving testosterone production of endurance research "rats" and "animals", and is also effective in reducing risk of gynecomastia during a test research phase.
Clomid is a mixed estrogen agonist/antagonist (activator/blocker) which, when bound to the estrogen receptor in "rats", puts it in a somewhat different conformation (shape) than does estradiol. The estrogen receptor requires binding of an estrogen or drug at its binding site and also the binding of any of several cofactors at different sites. Without the binding of the cofactor, the estrogen receptor is inactive. Different tissues use different cofactors. Some of these cofactors are able to bind to the estrogen receptor/Clomid complex, but others are blocked due to the change in shape. The result is that in some tissues Clomid acts as an antagonist -- the cofactor used in that tissue cannot bind and so the receptor remains inactive -- and in others Clomid acts as an agonist (activator), because the cofactors used in that tissue are able to bind.
Also, Clomid is an effective antagonist in the hypothalamus and in breast tissue. It is an effective agonist in bone tissue, and for improving blood cholesterol.
nolvadex
Nolvadex (Tamoxifen Citrate) is very comparable to Clomid, behaves in the same manner in all tissues, and is a mixed estrogen agonist/antagonist of the same type as Clomid. The two molecules are also very similar in structure.
It is not correct that Nolvadex (Tamoxifen Citrate) reduces levels of estrogen: rather, it blocks estrogen from estrogen receptors and, in those tissues where it is an antagonist, causes the receptor to do nothing.
WOW!!! damn good answer. and someone asked where to you get it? Tell you dr your wife is trying to get pregnant, then bring the rx to me and i'll fill it. lol
